参考文献/References:
[1] Maniatis T.A ubiquitin ligase complex essential for the NF-κB,Wnt/Wingless and Hedgehog signaling pathways[J].Genes Dev,1999,13:505-510.
[2] Ghosh S,May M J,Kopp EB.NF-κB and Rel proteins:evolutionarily conserved mediators of immune response[J].Ann Rev Immunol,1998,16:225-260.
[3] He SJ,Jin SZ,Liu SZ.Effect of whole-body X-irradiation on NF-κB activity in immune organs[J].J Radiat Res Radiat Proc,2000,18(4):278-283.
[4] Miyamoto S,Verma IM.Rel/NF-κB/IκB story[J].Adv Cancer Res,1995,66:255-292.
[5] Attar RM,Caamano J,Carraseo D,et al.Genetic approaches to study Rel/NF-κB/IκB function[J].Semin Cancer Biol,1997,8:93-101.
[6] Roulston A,Lin R,Beauparlant P,et al.Regalation of human immunodeficiency virus I and cytokine gene expression in myeloid cells by NF-κB/Rel transcription factors[J].Microbiol Rev,1995,59:481-505.
[7] Bex F,Gaynor RB.Regulation of gene expression by HTLV-I Tax protein[J].Methods,1998,16:83-94.
[8] Sha WC,Liou HC,Tuomanen E,et al.Targeted disruption of the p50 subunit of NF-kappaB leads to multifocal defects in immune responses[J].Cell,1995,80:321-330.
[9] Iotsova V,Caamano J,Loy J,et al.Osteoporosis inmice lacking NF-κB1 and NF-κB2[J].Nat Med,1997,3:1285-1289.
[10] Baichwal VR,Baeuerle PA.Activate NF-κB or die?[J]Curr Biol,1997,7:94-96.
[11] Sonenshein GE.NF-κB transcription factors and the control of apoptosis[J] Semin Cancer Biol,1997,8:113-119.
[12] Beg AA,Sha WC,Bronson RT,et al.Embrionic lethality and liver degeneration inmmice lacking the RelA component of NF-κB[J].Nature,1995,376:167-169.
[13] Beg AA,Baltimone D.An essential role for NF-κB in preventing TNF-α-induced cell death[J].Science,1996,274:782-784.
[14] Wang CC,Mayo MW,Baldwin Jr AS.TNF-and cancer therapy-induced apoptosis potentiation by inhibition of NF-κB[J].Science,1996,274:784-787.
[15] Grimm S,Bauer M,Baeuerle PA,et al.Bcl-2 down-regulation the activity of transcription factor NF-κB induced upon apoptosis[J].J Cell Biol,1996,34:13-23.
[16] Jung M,Zhang Y,Lee S,et al.Correction of radiation sensitivity in atxia telengiectasis by a truncated IκBα[J].Science,1995,268:1619-1621.
[17] Abbadie C,Kabrun N,Bouali F,et al.High levels of c-Rel expression are associated with progammed cell death in developing avian embryo and in bone marrow cells in vitro[J].Cell,1993,75:899-912.
[18] Bash J,Zong WX,Gelinas C.c-Rel arrests the proliferation of HeLa cells and affects critical regulators of the G1/S-phase transition[J].Mol Cell Biol,1997,17:6526-6536.
[19] Zong WX,Farrell M,Bash J,et al.v-Rel prevents apoptosis in transformed lymphoid cells and blocks TNF-α-induced cell death[J].Oncogene,1997,15:971-980.
[20] Tsunta L,Lee HJ,Masuda ES,et al.Cyclic AMP inhibits expression of the IL-2 gene through the nuclear factor of activated T cells (NF-AT) site and transfection of NF-AT cDNAs abrogates the sensitivity of EL-4 cells to cyclic AMP[J].J Immunol,1995,154:5255.
[21] Zwollo P,Rao S,Wallin JJ,et al.The transcription factor NF-κB/p50 interacts with the blk gene during B cell activation[J].J Biol Chem,1998,273:18647.
[22] Kang SM,Tran AAC,Grill M.NE-κB subunit regulation in non-transformed CD4+T lymphocytes[J].Science,1992,256:1452-1456.
[23] Wang CY,Cusack JC,Liu R,et al.Control of inducible chemoresistance:Enhanced anti-tumor therapy through increased apoptosis by inhibition of NF-κB[J].Nature Med,1999,5:412-417.
[24] Chen D,Rothenberg EV.Molecular basis for developmental changes in IL-2 gene inducibility[J].Mol Cell Biol,1993,133:228-237.
[25] Liu SZ,Zhang YC,Mu Y,et al.Thymocyte apoptosis in response to low dose radiation[J].Mutat Res,1996,358:185-191.
[26] Liu SZ.Multilevel mechanisms of stimulatory effect of low dose radiation on immunity[A].Sugahara T,Sagan L A and Aoyama T.Low dose irradiation and biological defense mechanisms[C].Elsevier Science Publishers,1992,225-232.
[27] Prasad AV,Mohan N,Chandrasekar B,et al.Activation of nuclear factor kappa B in human lymphoblastoid cells by low dose ionizing radiation[J].Radia Res,1994,138:367-372.
[28] Rice NR,Mackichan ML,Israel A.The precursor of NF-κB p50 has IκB-like functions[J].Cell,1992,78:773-785.
[29] Belich MP,Salmer A,Johnston LH,et al.TPL-2 kinase regulates the proteolytic processing of the human NF-κB1 precursor[J].Cene,1999,397:363-368.
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