[1].Long Non-coding RNAs in Response to Genotoxic Stress[J].国际放射医学核医学杂志,2016,40(2):133-138.[doi:10.3760/cma.j.issn.1673-4114.2016.02.010]
　Xiaoman Li,Dong Pan,Baoquan Zhao,et al.Long Non-coding RNAs in Response to Genotoxic Stress[J].International Journal of Radiation Medicine and Nuclear Medicine,2016,40(2):133-138.[doi:10.3760/cma.j.issn.1673-4114.2016.02.010]

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[1] Ciccia A, Elledge SJ. The DNA damage response:making it safe to play with knives[J]. Mol Cell, 2010, 40(2):179-204. DOI:10.1016/j. molcel. 2010. 09. 019.
[2] San Filippo J, Sung P, Klein H. Mechanism of eukaryotic homologous recombination[J]. Annu Rev Biochem, 2008, 77:229-257. DOI:10.1146/annurev. biochem. 77. 061306. 125255.
[3] Lieber MR. The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway[J]. Annu Rev Biochem, 2010, 79:181-211. DOI:10.1146/annurev. biochem. 052308. 093131.
[4] Polo SE, Blackford AN, Chapman JR, et al. Regulation of DNA-end resection by hnRNPU-like proteins promotes DNA double-strand break signaling and repair[J]. Mol Cell, 2012, 45(4):505-516. DOI:10.1016/j. molcel. 2011. 12. 035.
[5] Landau DA, Slack FJ. MicroRNAs in mutagenesis, genomic instability, and DNA repair[J]. Semin Oncol, 2011, 38(6):743-751. DOI:10.1053/j. seminoncol. 2011. 08. 003.
[6] Wan GH, Mathur R, Hu XX, et al. miRNA response to DNA damage[J]. Trends Biochem Sci, 2011, 36(9):478-484. DOI:10.1016/j. tibs. 2011. 06. 002.
[7] Wang X, Arai S, Song X, et al. Induced ncRNAs allosterically modify RNA-binding proteins in cis to inhibit transcription[J]. Nature, 2008, 454(720):126-130. DOI:10.1038/nature06992.
[8] Sharma V, Misteli T. Non-coding RNAs in DNA damage and repair[J]. FEBS Lett, 2013, 587(13):1832-1839. DOI:10.1016/j. febslet. 2013. 05. 006.
[9] Matsuoka S, Ballif BA, Smogorzewska A, et al. ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage[J]. Science, 2007, 316(5828):1160-1166. DOI:10.1126/science. 1140321.
[10] Toledo F, Wahl GM. Regulating the p53 pathway:in vitro hypotheses, in vivo veritas[J]. Nat Rev Cancer, 2006, 6(12):909-923. DOI:10.1038/nrc2012.
[11] Gibb EA, Vucic EA, Enfield KS, et al. Human cancer long non-coding RNA transcriptomes[J/OL]. PLoS One, 2011, 6(10):e25915[2015-11-24]. http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0025915. DOI:10.1371/journal.pone.0025915.
[12] Hung T, Wang Y, Lin MF, et al. Extensive and coordinated transcription of noncoding RNAs within cell-cycle promoters[J]. Nat Genet, 2011, 43(7):621-629. DOI:10.1038/ng. 848.
[13] Kitagawa M, Kitagawa K, Kotake Y, et al. Cell cycle regulation by long non-coding RNAs[J]. Cell Mol Life Sci, 2013, 70(24):4785-4794. DOI:10.1007/s00018-013-1423-0.
[14] Kurokawa R. Promoter-associated long noncoding RNAs repress transcription through a RNA binding protein TLS[J]. Adv Exp Med Biol, 2011, 722:196-208. DOI:10.1007/978-1-4614-0332-6_12.
[15] Liu X, Li D, Zhang W, et al. Long non-coding RNA gadd7 interacts with TDP-43 and regulates Cdk6 mRNA decay[J]. EMBO J, 2012, 31(23):4415-4427. DOI:10.1038/emboj. 2012. 292.
[16] Rossi MN, Antonangeli F. LncRNAs:new players in apoptosis control[J/OL]. Int J Cell Biol, 2014, 2014:473857[2015-11-24]. http://www.hindawi.com/journals/ijcb/2014/473857. DOI:10.1155/2014/473857.
[17] Huarte M, Guttman M, Feldser D, et al. A large intergenic noncoding RNA induced by p53 mediates global gene repression in the p53 response[J]. Cell, 2010, 142(3):409-419. DOI:10.1016/j.cell.2010.06.040.
[18] Zhang C, Peng G. Non-coding RNAs:an emerging player in DNA damage response[J]. Mutat Res Rev Mutat Res, 2015, 763:202-211. DOI:10.1016/j.mrrev.2014.11.003.
[19] Mok MT, Henderson BR. Three-dimensional imaging reveals the spatial separation of γH2AX-MDC1-53BP1 and RNF8-RNF168-BRCA1-A complexes at ionizing radiation-induced foci[J]. Radiother Oncol, 2012, 103(3):415-420. DOI:10.1016/j. radonc. 2012. 04. 009.
[20] Pfingsten JS, Goodrich KJ, Taabazuing C, et al. Mutually exclusive binding of telomerase RNA and DNA by Ku alters telomerase recruitment model[J]. Cell, 2012, 148(5):922-932. DOI:10.1016/j. cell. 2012. 01. 033.
[21] Lee YH, Kuo CY, Stark JM, et al. HP1 promotes tumor suppressor BRCA1 functions during the DNA damage response[J]. Nucleic Acids Res, 2013, 41(11):5784-5798. DOI:10.1093/nar/gkt231.
[22] Adamson B, Smogorzewska A, Sigoillot FD, et al. A genome-wide homologous recombination screen identifies the RNA-binding protein RBMX as a component of the DNA-damage response[J]. Nat Cell Biol, 2012, 14(3):318-328. DOI:10.1038/ncb2426.
[23] Nguyen D, Grenier St-Sauveur V, Bergeron D, et al. A Polyadenylation-Dependent 3’ end maturation pathway is required for the synthesis of the human telomerase RNA[J]. Cell Rep, 2015, 13(10):2244-2257. DOI:10.1016/j. celrep. 2015. 11. 003.
[24] Schoeftner S, Blasco MA. Chromatin regulation and non-coding RNAs at mammalian telomeres[J]. Semin Cell Dev Biol, 2010, 21(2):186-193. DOI:10.1016/j. semcdb. 2009. 09. 015.
[25] Wan G, Hu X, Liu Y, et al. A novel non-coding RNA lncRNA-JADE connects DNA damage signalling to histone H4 acetylation[J]. EMBO J, 2013, 32(21):2833-2847. DOI:10.1038/emboj. 2013. 221.
[26] Prensner JR, Chen W, Iyer MK, et al. PCAT-1, a long noncoding RNA, regulates BRCA2 and controls homologous recombination in cancer[J]. Cancer Res, 2014, 74(6):1651-1660. DOI:10.1158/0008-5472. CAN-13-3159.